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>   home   >   Products   >   Primary Antibodies   >   Antibody Collections   >   Mitochondrion   >   TFAM (Transcription Factor A, motochondrial) Antibody   

TFAM (Transcription Factor A, motochondrial) Antibody

Rabbit Polyclonal Antibody

     
  • WB - TFAM (Transcription Factor A, motochondrial) Antibody AN1293
    Western blot of HeLa lysate showing specific immunolabeling of the ~24 kDa TFAM protein.
  • SPECIFICATION
  • CITATIONS
  • PROTOCOLS
  • BACKGROUND
Product Information
Application
  • Applications Legend:
  • WB=Western Blot
  • IHC=Immunohistochemistry
  • IHC-P=Immunohistochemistry (Paraffin-embedded Sections)
  • IHC-F=Immunohistochemistry (Frozen Sections)
  • IF=Immunofluorescence
  • FC=Flow Cytopmetry
  • IC=Immunochemistry
  • ICC=Immunocytochemistry
  • E=ELISA
  • IP=Immunoprecipitation
  • DB=Dot Blot
  • CHIP=Chromatin Immunoprecipitation
  • FA=Fluorescence Assay
  • IEM=Immunoelectronmicroscopy
  • EIA=Enzyme Immunoassay
WB
Primary Accession Q00059
Reactivity Human
Host Rabbit
Clonality Polyclonal
Calculated MW 29097 Da
Additional Information
Gene ID 7019
Gene Name TFAM
Target/Specificity Native recombinant human TFAM protein with c-terminal 6-his tag
Dilution WB~~ 1:2000
Format Neat Serum
StorageMaintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles.
PrecautionsTFAM (Transcription Factor A, motochondrial) Antibody is for research use only and not for use in diagnostic or therapeutic procedures.
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Research Areas
Citations (0)

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Background

Mitochondrial Transcription Factor A (TFAM) is a key activator of mitochondrial (mt) DNA transcription as well as a participant in mitochondrial genome replication. mtDNA is highly susceptible to oxidative stress leading to mitochondrial dysfunction. Overexpression of TFAM has been implicated in the amelioration of age dependent impairment of brain functions through the prevention of oxidative stress and mitochondrial dysfunction in microglia (Hayashi et al., 2008). More recently, TFAM overexpression has been shown to potentially reduce oxidative stress in motor neurons and delay onset of amyotrophic lateral sclerosis (ALS) in ALS model mice (Morimoto et al., 2012).

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$ 310.00
Cat# AN1293
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