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SFRS16 Antibody (Center)
Affinity Purified Rabbit Polyclonal Antibody (Pab)
- SPECIFICATION
- CITATIONS
- PROTOCOLS
- BACKGROUND
Application 
| FC, WB, E |
|---|---|
| Primary Accession | Q8N2M8 |
| Other Accession | Q5HZB6, Q8CFC7, A0JNI5, NP_008987.2 |
| Reactivity | Human |
| Predicted | Bovine, Mouse, Rat |
| Host | Rabbit |
| Clonality | Polyclonal |
| Isotype | Rabbit IgG |
| Calculated MW | 77162 Da |
| Antigen Region | 254-283 aa |
| Gene ID | 11129 |
|---|---|
| Other Names | CLK4-associating serine/arginine rich protein, Splicing factor, arginine/serine-rich 16, Suppressor of white-apricot homolog 2, CLASRP, SFRS16, SWAP2 |
| Target/Specificity | This SFRS16 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 254-283 amino acids from the Central region of human SFRS16. |
| Dilution | WB~~1:1000 FC~~1:10~50 |
| Format | Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification. |
| Storage | Maintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles. |
| Precautions | SFRS16 Antibody (Center) is for research use only and not for use in diagnostic or therapeutic procedures. |
| Name | CLASRP |
|---|---|
| Synonyms | SFRS16, SWAP2 |
| Function | Probably functions as an alternative splicing regulator. May regulate the mRNA splicing of genes such as CLK1. May act by regulating members of the CLK kinase family (By similarity). |
| Cellular Location | Nucleus. |
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Provided below are standard protocols that you may find useful for product applications.
Background
SFRS16 is probably functions as an alternative splicing regulator. SFRS16 may regulate the mRNA splicing of genes such as CLK1. This may act by regulating members of the CLK kinase family.
References
Hosgood, H.D. III, et al. Occup Environ Med 66(12):848-853(2009)
Sugiyama, N., et al. Mol. Cell Proteomics 6(6):1103-1109(2007)
Olsen, J.V., et al. Cell 127(3):635-648(2006)
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