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FGF9 Antibody (N-term) Blocking peptide
Synthetic peptide
- SPECIFICATION
- CITATIONS
- PROTOCOLS
- BACKGROUND
| Primary Accession | P31371 |
|---|---|
| Other Accession | NP_002001.1 |
| Clone Names | 91009102 |
| Gene ID | 2254 |
|---|---|
| Other Names | Fibroblast growth factor 9, FGF-9, Glia-activating factor, GAF, Heparin-binding growth factor 9, HBGF-9, FGF9 |
| Format | Peptides are lyophilized in a solid powder format. Peptides can be reconstituted in solution using the appropriate buffer as needed. |
| Storage | Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C. |
| Precautions | This product is for research use only. Not for use in diagnostic or therapeutic procedures. |
| Name | FGF9 |
|---|---|
| Function | Plays an important role in the regulation of embryonic development, cell proliferation, cell differentiation and cell migration. May have a role in glial cell growth and differentiation during development, gliosis during repair and regeneration of brain tissue after damage, differentiation and survival of neuronal cells, and growth stimulation of glial tumors. |
| Cellular Location | Secreted. |
| Tissue Location | Glial cells. |
Thousands of laboratories across the world have published research that depended on the performance of antibodies from Abcepta to advance their research. Check out links to articles that cite our products in major peer-reviewed journals, organized by research category.
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Provided below are standard protocols that you may find useful for product applications.
Background
The protein encoded by this gene is a member of thefibroblast growth factor (FGF) family. FGF family members possessbroad mitogenic and cell survival activities, and are involved in avariety of biological processes, including embryonic development,cell growth, morphogenesis, tissue repair, tumor growth andinvasion. This protein was isolated as a secreted factor thatexhibits a growth-stimulating effect on cultured glial cells. Innervous system, this protein is produced mainly by neurons and maybe important for glial cell development. Expression of the mousehomolog of this gene was found to be dependent on Sonic hedgehog(Shh) signaling. Mice lacking the homolog gene displayed amale-to-female sex reversal phenotype, which suggested a role intesticular embryogenesis.
References
Jugessur, A., et al. PLoS ONE 5 (7), E11493 (2010) :Yokoyama, K., et al. Nephron Clin Pract 115 (4), C237-C243 (2010) :Kalinina, J., et al. Mol. Cell. Biol. 29(17):4663-4678(2009)Marroni, F., et al. Circ Cardiovasc Genet 2(4):322-328(2009)Wu, X.L., et al. Am. J. Hum. Genet. 85(1):53-63(2009)
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