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MIPEP Antibody (N-term)
Purified Rabbit Polyclonal Antibody (Pab)
- SPECIFICATION
- CITATIONS
- PROTOCOLS
- BACKGROUND
Application 
| IHC-P, WB, E |
|---|---|
| Primary Accession | Q99797 |
| Reactivity | Human |
| Host | Rabbit |
| Clonality | Polyclonal |
| Isotype | Rabbit IgG |
| Calculated MW | 80641 Da |
| Antigen Region | 180-209 aa |
| Gene ID | 4285 |
|---|---|
| Other Names | Mitochondrial intermediate peptidase, MIP, MIPEP, MIP |
| Target/Specificity | This MIPEP antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 180-209 amino acids from the N-terminal region of human MIPEP. |
| Dilution | IHC-P~~1:10~50 WB~~1:1000 E~~Use at an assay dependent concentration. |
| Format | Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is prepared by Saturated Ammonium Sulfate (SAS) precipitation followed by dialysis against PBS. |
| Storage | Maintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles. |
| Precautions | MIPEP Antibody (N-term) is for research use only and not for use in diagnostic or therapeutic procedures. |
| Name | MIPEP |
|---|---|
| Synonyms | MIP |
| Function | Cleaves proteins, imported into the mitochondrion, to their mature size. |
| Cellular Location | Mitochondrion matrix. |
Thousands of laboratories across the world have published research that depended on the performance of antibodies from Abcepta to advance their research. Check out links to articles that cite our products in major peer-reviewed journals, organized by research category.
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Provided below are standard protocols that you may find useful for product applications.
Background
MIPEP performs the final step in processing a specific class of nuclear-encoded proteins targeted to the mitochondrial matrix or inner membrane. This protein is primarily involved in the maturation of oxidative phosphorylation (OXPHOS)-related proteins. This protein may contribute to the functional effects of frataxin deficiency and the clinical manifestations of Friedreich ataxia.
References
Chew A., Genomics 40:493-496(1997).
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